Online ACLS Course

chapter 16 : Stable Tachycardia

Tachycardia is when the heart rate or rhythm is too fast (>100 beats/min). Increase in heart rate causes less blood to be pumped through the systemic and pulmonary systems. Low blood flow will cause less oxygen to flow to the heart and brain and less oxygen to the heart can lead to Ischemia and MI.   In stable tachycardia the patient is stable and no significant signs and symptoms are present.

Video Demonstration

Stable Tachycardia

The rhythms for stable tachycardia include:

Atrial fibrillation is when the heart beats do not occur at the same intervals.  It is known as the quivering of the muscles and involves both the atriums of the heart.

Atrial flutter is abnormal heart rhythm causing fast irregular heartbeat.  Starts in the atrium and can lead to atrial fibrillation. Usually has a ‘saw-toothed’ appearance.

Sinus tachycardia is when the rate of impulse from the SA node is elevated.

AV nodal reentry is a heart rhythm that is seen due to the malfunctioning of the AV node. It is the most common type of SVT

Supraventricular Tachycardia (SVT) is a rapid and narrow heartbeat that starts in the atria or AV node.

Monomorphic VT is heart rate of >150 bpm but QRS complexes look the same.

Polymorphic VT is when different areas in the ventricles fire fast, uncoordinated impulses.

Multifocal Atrial Tachycardia is when the atrial pacemaker rhythm is deranged and causes the ventricular rate to be greater than 100 bpm. This rhythm causes the P waves to differ in size, shape and direction.

Scenario: You are the night nurse on duty and a  40 year old woman is complaining of palpitations.  She mentions that she has a history of recurrent heart rates and she feels she’s having a heart attack.

Assessment:

1. The patient is responsive and is breathing
2. Attach the patient on a monitor and identify tachycardia (>100 bpm)
3. Call the doctor on duty

Interventions:

1. Maintain airway if needed
2. Help with breathing and give oxygen if hypoxemic and monitor O2 saturation
3. Monitor BP and HR and conduct a 12-lead ECG and diagnose
4. Check for persistent tachyarrhythmia

Management: at the hospital

1. 1. If persistent tachyarrhythmia initiate synchronized cadioversion
      1. Narrow regular 50-100 j
      2. Narrow irregular 120-200 J biphasic and 200 J monophasic
      3. Wide regular 100 J
      4. Wide irregular defibrillation dose

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1. Administer Adenosine IV dose 6mg rapid IV push with NS flush and 12 mg for second dose
2. If not persistent tachyarrythmia and if wide QRS ≥12 seconds then obtain IV access and 12-lead ECG
3. Administer Adenosine if monomorphic
4. Administer antiarrhythmic infusion (Procaainaminde, Amiodorone, Sotalol)
5. If not wide QRS consider: vagal maneuvers, adenosine, beta blockers, calcium channel blockers

The following is an algorithm showing management of stable tachycardia in detail.

If a patient shows stable tachycardia they will not present with hypotension, shock, ischemic chest pain, AMS or heart failure. In a case when the patient is stable and has regular rhythm, consider administering adenosine and attempting to do vagal maneuvers.

Vagal maneuvers and administration of adenosine is the initial first line intervention for narrow complex stable tachycardias. These maneuvers initiate baroreceptors in the aortic arch and the internal carotid arteries, which then cause stimulation of the Vagus nerve which releases acetylcholine. The acetylcholine reduces AV node conduction and that eventually slows down the heart rate.

Types of Vagal maneuvers include:

If regular SVT is not fixed by adenosine or vagal maneuvers then the best option is to seek expert advice.

Learning Outcomes:
You have completed Chapter XII. Now you should be able to:

1. Recognize the types of ECG rhythms associated with Tachycardia
2. Apply the Adult Tachycardia Algorithm in Stable Tachycardia
3. Understand vagal maneuvers